Download An Atlas of Gastroenterology: A Guide to Diagnosis and by Cyrus R. Kapadia PDF

By Cyrus R. Kapadia

Going past mere analysis, An Atlas of Gastroenterology covers every little thing from swallowing problems to remedy of jaundiced and immuno-compromised sufferers. Tables, illustrations, and case histories placed the data at your fingertips. prime specialists mix lucid textual content with large illustrations to interpret the techniques wanted for an organization clutch of the subject. insurance contains swallowing problems, heartburn, dyspepsia, diarrhea, irritable bowel syndrome, colonic polyps and colon melanoma, pancreatitis, pancreatic melanoma, the jaundiced sufferer and the immuno-compromised sufferer. released mostly for family members and first care physicians An Atlas of Gastroenterology describes all elements of the analysis and therapy of gastrointestinal ailment.

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Extra resources for An Atlas of Gastroenterology: A Guide to Diagnosis and Differential Diagnosis

Sample text

He completed his master’s degree and worked for a bank. As a rule he was free of pain and passed one to two bowel movements each day. Occasionally he would experience some diarrhea accompanied by a little abdominal pain. He required short courses of oral corticosteroids on two occasions during the 5-year period. He was never hospitalized for any of these mild recurrences. He now presents with bloating and colicky abdominal pain of 5 or 6 days’ duration. A day prior to admission he began vomiting copious amounts of bilious material.

Pathology The mucosal lesion in celiac disease occurs in three stages: (1) Stage I Increase in the number of intraepithelial lymphocytes, many of which are γ/δ T cells. This is followed by an increase in lamina propria lymphocytes. 20 Prolamines of cereals Amino acids Wheat: gliadins (α,β,γ,ω) Barley: hordeins Rye: secalins Oats: avenins Corn and rice: (2) Stage II glutamine > 35%; proline > 15% glutamine > 35%; proline > 15% glutamine > 35%; proline > 15% glutamine fairly high; proline low glutamine low; proline very little or none Crypt hyperplasia.

The mechanisms of intestinal malabsorption of fat and cobalamin due to bacterial overgrowth are described in Chapter 6. For the present, it is sufficient to say that these bacteria, not normally present in the upper jejunum, which is normally almost sterile, result in the deconjugation and dehydroxylation of bile acids. As a consequence, micelle formation does not occur and steatorrhea is the result. Also, these bacteria avidly bind the intrinsic factor–cobalamin complex, thus making it unavailable for attachment to the special receptors present in the ileum.

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